File:Myocardial-contractility-in-the-stress-echo-lab-from-pathophysiological-toy-to-clinical-tool-1476-7120-11-41-S2.ogv
Myocardial-contractility-in-the-stress-echo-lab-from-pathophysiological-toy-to-clinical-tool-1476-7120-11-41-S2.ogv (Ogg multiplexed audio/video file, Theora/Vorbis, length 1 min 31 s, 648 × 486 pixels, 645 kbps overall, file size: 7.01 MB)
Captions
Summary
[edit]DescriptionMyocardial-contractility-in-the-stress-echo-lab-from-pathophysiological-toy-to-clinical-tool-1476-7120-11-41-S2.ogv |
English: Dilated Cardiomyopathy: The sarcomere At each cycle of heartbeat, the heart cell contracts (shortens) at systole and relaxes at diastole. Under stress, both the speed and the intensity of the movement are increased. In the DCM heart, both the structure and the activity are impaired: the mitochondrion is enlarged, due to the excess ATP request, and the fibers are not as compact and aligned as in the healthy fiber. The relaxation phase is never complete, resulting in the typical ECG track. Under stress the situation is worsened: the heart fibers, unable to completely de-contract, try to compensate by even stronger contraction, in a forward loop that worsens with time, and can lead to heart failure. (The full quality video can be accessed at this link: https://vimeo.com/79085938). |
||
Date | |||
Source | Bombardini T, Zoppe M, Ciampi Q, Cortigiani L, Agricola E, Salvadori S, Loni T, Pratali L, Picano E (2013). "Myocardial contractility in the stress echo lab: from pathophysiological toy to clinical tool". Cardiovascular Ultrasound. DOI:10.1186/1476-7120-11-41. PMID 24246005. PMC: 3875530. | ||
Author | Bombardini T, Zoppe M, Ciampi Q, Cortigiani L, Agricola E, Salvadori S, Loni T, Pratali L, Picano E | ||
Permission (Reusing this file) |
This file is licensed under the Creative Commons Attribution 2.0 Generic license.
|
||
Provenance InfoField |
|
File history
Click on a date/time to view the file as it appeared at that time.
Date/Time | Thumbnail | Dimensions | User | Comment | |
---|---|---|---|---|---|
current | 03:17, 1 January 2014 | 1 min 31 s, 648 × 486 (7.01 MB) | Open Access Media Importer Bot (talk | contribs) | Automatically uploaded media file from Open Access source. Please report problems or suggestions here. |
You cannot overwrite this file.
File usage on Commons
There are no pages that use this file.
Transcode status
Update transcode statusMetadata
This file contains additional information such as Exif metadata which may have been added by the digital camera, scanner, or software program used to create or digitize it. If the file has been modified from its original state, some details such as the timestamp may not fully reflect those of the original file. The timestamp is only as accurate as the clock in the camera, and it may be completely wrong.
Short title | Additional file 2 |
---|---|
Author | Bombardini T, Zoppe M, Ciampi Q, Cortigiani L, Agricola E, Salvadori S, Loni T, Pratali L, Picano E |
Usage terms | http://creativecommons.org/licenses/by/2.0/ |
Image title | Dilated Cardiomyopathy: The sarcomere At each cycle of heartbeat, the heart cell contracts (shortens) at systole and relaxes at diastole. Under stress, both the speed and the intensity of the movement are increased. In the DCM heart, both the structure and the activity are impaired: the mitochondrion is enlarged, due to the excess ATP request, and the fibers are not as compact and aligned as in the healthy fiber. The relaxation phase is never complete, resulting in the typical ECG track. Under stress the situation is worsened: the heart fibers, unable to completely de-contract, try to compensate by even stronger contraction, in a forward loop that worsens with time, and can lead to heart failure. (The full quality video can be accessed at this link: https://vimeo.com/79085938). |
Software used | |
Date and time of digitizing | 2013 |