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H7N2 virus
Summary[edit]
| Description |
English: Figure 1. Zoonotic influenza pathology is linked to host biology. H5N1, H7N9, and H9N2 avian influenza (AI) viruses causing varying degrees of clinical manifestations across different species is shown against different host and their sialic acid properties. AI viral hemagglutinin proteins selectively and preferentially bind to sialic residues with either an α-2,3-Gal or α-2,6-Gal terminating sequence on the host cell surface to allow for viral fusion and entry. This represents viral fitness and is a potential mechanism underlying the degree of disease severity and susceptibility in the different hosts. |
| Date | |
| Source | https://www.frontiersin.org/journals/immunology/articles/10.3389/fimmu.2018.01812/full |
| Author | William S. J. Horman,Thi H. O. Nguyen,Katherine Kedziers,Andrew G. D. Bean,Daniel S. Layton |
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| Date/Time | Thumbnail | Dimensions | User | Comment | |
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| current | 22:25, 29 February 2024 | | 250 × 450 (23 KB) | Ozzie10aaaa (talk | contribs) | resize |
| 22:18, 29 February 2024 |  | 148 × 240 (17 KB) | Ozzie10aaaa (talk | contribs) | Uploaded a work by William S. J. Horman,Thi H. O. Nguyen,Katherine Kedziers,Andrew G. D. Bean,Daniel S. Layton from https://www.frontiersin.org/journals/immunology/articles/10.3389/fimmu.2018.01812/full with UploadWizard |
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