File:The diagnosis and treatment of diseases of women (1907) (14598031299).jpg

Identifier: diagnosistreatmecros (find matches)
Title: The diagnosis and treatment of diseases of women
Year: 1907 (1900s)
Authors: Crossen, Harry Sturgeon, 1869-
Subjects: Genital Diseases, Female Gynecology Gynecology Women Generative organs, Female
Publisher: St. Louis : Mosby
Contributing Library: Francis A. Countway Library of Medicine
Digitizing Sponsor: Open Knowledge Commons and Harvard Medical School

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tion of the mucosa usually give rise toa hemorrhagic tendency. The hypertrophy or hyperplasia may progress to such 568 DISEASES OF THE UTERUS an extent that the mucosa becomes many times its usual thickness. Whenthe hyperplasia is so marked, it usually takes place unevenly, so that thesurface is rough and nodular, giving rise to the name fungous endometritis.The normal endometrium is shown in Fig. 568. Chronic endometritis of thefungous form is shown in Figs. 569, 570. In this condition the hemorrhagictendency is a marked feature, hence the name hemorrhagic endometritis.In some cases the masses project out from the surface and become pedi-culated and give rise to polypi. This condition is known also as poly-poid endometritis. The gland-ducts become obstructed and retentioncysts are thus formed. In the fungus and polypoid form of endome-tritis, the interstitial tissue in the endometrium undergoes decided increaseand hence the condition is sometimes designated interstitial endometritis,
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Fig. 568. A Normal Uterus divided from in front, showing tlieSmoothness of the Endometrium and also its relative Thickness.,(Cullen—Cancer of the Uterus.) in contradistinction to glandular endometritis, in whFch there is marked pro-liferation of the glands without corresponding increase in the connective tissue.After a long time the cellular infiltration largely disappears, new connective tissuetaking its place, and this connective tissue contracts as the infiltration-cells betweenthe fibers disappear. The glands are thus injuriously pressed upon and begin toundergo pressure-atrophy, their ducts are obstructed and cystic dilatation takesplace. This process becomes more and more marked until there is great destruc-tion of gland tissue and the condition passes into sclerosis of the uterus, describedlater, in which little remains of the mucosa but scar-tissue. The change from ordi-nary chronic endometritis to the condition of sclerosis takes several years, except SYMPTOMS OF CHRONIC IN

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