File:Refraction and motility of the eye, with chapters on color blindness and the field of vision (1920) (14767845984).jpg

Identifier: refractionmotili00alge (find matches)
Title: Refraction and motility of the eye, with chapters on color blindness and the field of vision
Year: 1920 (1920s)
Authors: Alger, Ellice Murdoch, 1870- (from old catalog)
Subjects: Eye Eye
Publisher: Philadelphia, F. A. Davis petc., etc.)
Contributing Library: The Library of Congress
Digitizing Sponsor: The Library of Congress

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Fig. 115. are any objective changes it is possible to demonstrate anenlargement of the blind spot, and later a characteristicnotching of the field in the upper nasal quadrant. 346 REFRACTION AND MOTILITY OF THE EYE. This narrowing occurs long before there is any reduc-tion in central vision and can be detected with a coloredobject long before it is evident with a white one. Eedue-tion in the field for colors is always followed by reductionfor white unless the progress is stopped. Pig. 114. Simple atrophy, showing concentric reduc- L. E. R. E.
Text Appearing After Image:
w Fig. 116. tion, central vision reduced, and the individual becomingcolor-blind early first for red, then green and last for blue. Fig. 115. Eetinitis pigmentosa. Central vision maybe normal in good light. Reduction of field is concentric,begins very early and increases enormously when the illum-ination is slightly reduced. Blue color sense is lost first. Fig. 116. Eight eye shows central scotoma from mac-ular chorioidal changes. Similar conditions at times result-from high myopia, syphilis, and senile degeneration. Thefield outside is normal for white and colors, but is very THE FIELD OF VISION. 347 difficult to map out owing to inability of patient to fix andkeep the eye still with a blind macula. Left eye shows typical field in retrobulbar neuritisfrom tobacco, alcohol, etc. Central vision reduced andbetter in dim light. Field for white normal at first, but acentral scotoma for red and green, and finally for white. Fiir. 11T. Eight eve shows sector deficiencv due to L. E. R. E. KA

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