File:Parasite150072-fig1 Role of neutrophils in rodent amebic liver abscess.png

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English: Figure 1 of paper
The initial contact between the lectin and neutrophil receptors promotes CR3 expression by neutrophils, and then the interaction of this molecule with iC3b induces neutrophil degranulation. It is likely that neutrophils are activated due to the recognition by TLR-2 and TLR-4 of lipopeptidophosphoglycan (LPPG) from Entamoeba histolytica. Activated neutrophils undergo an “oxidative burst” that results in an increased production of superoxide (O2−) by the NADPH-oxidase system. Superoxide dismutase (SOD) rapidly converts O2− to hydrogen peroxide (H2O2, a highly oxidizing agent), and to the substrate of MPO for the formation of hypochlorous acid (HOCl). During this oxidative burst, neutrophils express high levels of iNOS, causing an increase in •NO. Another powerful oxidant is peroxynitrite, formed by the reaction of •NO and O2•−.
Date
Source (2016). "A review of the proposed role of neutrophils in rodent amebic liver abscess models". Parasite 23: 6. DOI:10.1051/parasite/2016006. ISSN 1776-1042.
Author Rafael Campos-Rodríguez, Manuel Gutiérrez-Meza, Rosa Adriana Jarillo-Luna, María Elisa Drago-Serrano, Edgar Abarca-Rojano, Javier Ventura-Juárez, Luz María Cárdenas-Jaramillo and Judith Pacheco-Yepez

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Parasite
Parasite
This file was published in the scientific journal Parasite. Their website states that all content of the journal including and after 2013 is published under the Creative Commons Attribution 4.0 license.

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