File:Manual of pathological anatomy (1875) (14781469972).jpg

Identifier: manualofpatholog00jone (find matches)
Title: Manual of pathological anatomy
Year: 1875 (1870s)
Authors: Jones, C. Handfield, (Charles Handfield), 1819-1890 Sieveking, Edward H. (Edward Henry), 1816-1904 Payne, Joseph Frank, 1840-1910, ed
Subjects: Anatomy, Pathological Anatomy Pathology
Publisher: London : Churchill
Contributing Library: Francis A. Countway Library of Medicine
Digitizing Sponsor: Open Knowledge Commons and Harvard Medical School

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gether into an irregular mass. Theanaemic condition of lardaceous organs is thus explained bothby the impermeability of the vessels and by the swelling of thetissue elements, which, pressing on the vessels, hinder the supplyof blood. Distribution of Lardaceous Degeneration.—The organs most fre-quently affected are the spleen, liver, kidneys, and lymphatic glands. Second in order offrequency must (accordingto Yagner) be placed themucous membrane of theintestinal canal, the supra-renal capsules, and theomentum. More rarely(and seldom or never with-out simultaneous affectionof some of those organsjust mentioned) the samechange is found in themucous membrane of otherparts of the digestive tract,the pancreas, the mucousmembranes of the urinaryorgans, the generative or-gans, the heart, thyroidbody, lungs, and bronchi;as also, it is said, in bloodclots.Causes of Lardaceous Degeneration.—This affection almost alwaysoccurs as the sequel of some constitutional disease or exhausting Fig. 15.
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Malpig-hian tufts, and a small artery of thekidney affected with lardaceous degeneration,(After Rindfleisch.) powerful but momentary action of the acid results, which g-encrally (though it some-times fails) produces a pennanent black or purplith colour in those parts previouslystained by iodine. DEGENERATIONS. 129 local affection; very rarely it appears to be idiopathic, or at leastthe primary disease cannot be traced. By far the most frequentdetermining cause is some long-continued suppuration, such asaccompanies necrosis or caries of bone, chronic abscess, jointdisease, &c. In the same way it may follow pulmonary phthisiswhere there is much inflammatory destruction of the lung, dilata-tion of the bronchi with suppuration, empyema, chronic intestinalidceration, pyelitis, cystitis, and, it is said, chrooic ulceration ofthe skin. Syphilis is a very frequent precursor of waxy degenera-tion, and this usually when it takes the form of bone disease orchronic ulceration; but by no means

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