File:Fddsv-02-1015545-g001 (1).jpg

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Captions

Captions

transthyretin cardiac amyloidosis

Summary

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Description
English: FIGURE 1. Overview of the development of hereditary transthyretin cardiac amyloidosis. The liver synthesizes and excretes transthyretin (TTR) into the circulation to function as a transport complex. Mutations in TTR (e.g., Val122Iso, V122I) increases the propensity of the TTR tetrameric complex to dissociate into monomers. TTR monomers can by taken up by the myocardium, aggregated, and form amyloid fibrils. The deposition of amyloid fibrils in the heart may lead to restrictive cardiomyopathy.
Date
Source https://www.frontiersin.org/articles/10.3389/fddsv.2022.1015545/full
Author Marcus A. C. Williams,Bairavi Shankar,Joban Vaishnav,Mark J. Ranek

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current21:46, 24 February 2024Thumbnail for version as of 21:46, 24 February 2024761 × 500 (59 KB)Ozzie10aaaa (talk | contribs)Uploaded a work by Marcus A. C. Williams,Bairavi Shankar,Joban Vaishnav,Mark J. Ranek from https://www.frontiersin.org/articles/10.3389/fddsv.2022.1015545/full with UploadWizard

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