File:A treatise on the nervous diseases of children - for physicians and students (1895) (14784114865).jpg

Identifier: treatiseonnervou00sach (find matches)
Title: A treatise on the nervous diseases of children : for physicians and students
Year: 1895 (1890s)
Authors: Sachs, Bernard, 1858-1944
Subjects: Nervous System Diseases Pediatrics
Publisher: New York : William Wood
Contributing Library: Francis A. Countway Library of Medicine
Digitizing Sponsor: Open Knowledge Commons and Harvard Medical School

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or, to the absence of convulsions, andto the visual troubles. But a cerebral condition—a meningo-encephalitis—due to difficulties at birth, may be associatedwith a defective development of the pyramidal tract. Thisis the interpretation which I now give to the following case,which puzzled me for a long time, and presented a numberof anomalous features.f The patient, a boy, one year of age at the time of death, was born of ahealthy mother (primipara) after a dry labor of forty-eight hours. The child * For a fuller description of the anatomical condition, the reader is referred to thechapter on Diseases due to Defective Development of the Brain. t This case was published in full in the New York Medical Journal for May 2, 1891. 400 THE NERVOUS DISEASES OF CHILDREN. was asphyxiated. From the first day to the age of six and a half months itpassed through innumerable convulsive seizures of a very extreme order. (SeeFig. 38.) It presented in addition : Retarded mental development; spastic
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/(/f/ICOOU/jiO A^J> Fig. 98.—Section through Motor Cortex in Case of Spastic Paraplegia, a, space be-tween the cortex and pia; b, blood-vessels in transverse and longitudinal sections,showing infiltration of their walls ; c, altered cells with pericellular spaces ; /, the pia,thickened and infiltrated, sending projection downward between two convolutions. paraplegia (upper extremities less paralyzed, but somewhat rigid); increaseddeep reflexes (patellar, clonus, etc.), and convergent strabismus. The epilepticseizures were lessened by treatment, but otherwise the condition remainedunchanged. The child died of an acute infectious disease of a doubtfulcharacter. (So reported from the Babies Hospital.) HEREDITARY DISEASES OE THE SPINAL CORD. 4OI On post-mortem examination I found distinct evidencesof a wide-spread chronic meningo-encephalitis (probably dueto meningeal hemorrhage at birth, see Fig. 98), and in ad-dition, what I supposed at the time was a secondary de-generation in the

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