File:40478 2019 730 Fig1 HTML.webp
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Multiple system atrophy
Summary
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| Description |
English: Alpha-synuclein in the pathogenesis of MSA. a Putative mechanisms leading to α-syn intracellular accumulation, including SNCA CpG islands hypomethylation, transcription factors, lncRNAs, miRNAs, impaired lysosomal and proteasomal machineries. b Neuron-oligodendrocyte interaction mechanisms potentially involved in α-syn accumulation: oligodendroglial α-syn uptake from surrounding neurons and extracellular environment through endocytosis and passive transmembrane diffusion. Met = methyl group; TF = transcription factor; miRNAs = microRNAs; lncRNAs = long-non-coding RNAs |
| Date | |
| Source | https://actaneurocomms.biomedcentral.com/articles/10.1186/s40478-019-0730-6 |
| Author | Giacomo Monzio Compagnoni & Alessio Di Fonzo |
Licensing
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This file is licensed under the Creative Commons Attribution 4.0 International license.
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| Date/Time | Thumbnail | Dimensions | User | Comment | |
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| current | 21:07, 2 April 2024 | | 685 × 396 (24 KB) | Ozzie10aaaa (talk | contribs) | Uploaded a work by Giacomo Monzio Compagnoni & Alessio Di Fonzo from https://actaneurocomms.biomedcentral.com/articles/10.1186/s40478-019-0730-6 with UploadWizard |
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